自然杂志 ›› 2020, Vol. 42 ›› Issue (6): 471-474.doi: 10.3969/j.issn.0253-9608.2020.06.006
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张泰瑜，冯红
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ZHANG Taiyu, FENG Hong
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摘要： 帕金森病是第二大神经退行性疾病，目前发病机制尚不明确且无法治愈。抗增殖蛋白2参与维持线粒体形态和功能的稳定、凋亡等多种生物学进程。文章主要阐述抗增殖蛋白2与帕金森病之间的联系，进而探讨抗增殖蛋白2在帕金森病发病 线粒体机制中的作用。
关键词: 抗增殖蛋白2, 帕金森病, 线粒体机制
Abstract: Parkinson’s disease, the second largest neurodegenerative disease, cannot be cured. Its pathogenesis is currently unclear. Prohibitin 2 participates in various biological processes such as apoptosis and maintaining the stability of mitochondrial morphology and function. The present review focuses on the relationship between prohibitin 2 and Parkinson’s disease, and then explores the role of prohibitin 2 in the mechanism of mitochondria in the pathogenesis of Parkinson’s disease.
Key words: , prohibitin 2, Parkinson’s disease, mitochondrial mechanism
张泰瑜, 冯红. 抗增殖蛋白2在帕金森病发病线粒体机制中的作用[J]. 自然杂志, 2020, 42(6): 471-474.
ZHANG Taiyu, FENG Hong. Influence of prohibitin 2 in mitochondrial mechanism of Parkinson’s disease[J]. Chinese Journal of Nature, 2020, 42(6): 471-474.
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链接本文: https://www.nature.shu.edu.cn/CN/10.3969/j.issn.0253-9608.2020.06.006
https://www.nature.shu.edu.cn/CN/Y2020/V42/I6/471
